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Actavis is usually a NSAID that inhibits both of those isoforms of COX enzyme, a crucial enzyme linked to the inflammatory cascade. COX-1 enzyme is actually a constitutive enzyme linked to prostacyclin generation and protective features of gastric mucosa whereas COX-2 is an inducible enzyme linked to the production of inflammatory mediators in response to inflammatory stimuli. Actavis shows additional selectivity in the direction of COX-2 (IC50 of 0.77uM) than COX-one (IC50 of >100uM), which encourages its gastric tolerance when compared with other NSAIDs.
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Atorvastatin is very sure to plasma proteins and about 98% from the administered dose is located in a sure sort.
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